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Field notes · chapter 51

The Reproductive System

pp. 1133–1154 · Raven Part VII

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Table 51.1 — Mammalian reproductive hormones (memorize this one)

10 rows
HormoneSourceJob in the MALEJob in the FEMALE
GnRHHypothalamusMakes the anterior pituitary release FSH and LHSame — makes the anterior pituitary release FSH and LH
FSHAnterior pituitaryStimulates Sertoli cells → supports spermatogenesisStimulates follicle growth and estradiol secretion
LHAnterior pituitaryStimulates Leydig cells → testosterone secretionTriggers ovulation, converts follicle → corpus luteum, drives its estradiol/progesterone
TestosteroneLeydig cellsMaintains secondary sex characteristics, accessory organs, spermatogenesis; inhibits LH and GnRH
InhibinSertoli cellsSuppresses FSH only
EstradiolGranulosa cells, then corpus luteumFemale secondary sex characteristics; builds the endometrium each month; triggers the LH surge midcycle
ProgesteroneCorpus luteumCompletes uterine preparation for pregnancy; helps maintain secondary sex characteristics
hCGEmbryonic chorionLH-like; maintains the corpus luteum in early pregnancy; detected by pregnancy tests
OxytocinPosterior pituitaryUterine contraction and milk-ejection reflex
ProlactinAnterior pituitaryMilk production

The 28-day cycle — ovarian and uterine, side by side

5 rows
DaysOvarian cycleUterine cycleHormonesKey event
0–5Follicular phase beginsMenstruationEstradiol and progesterone at their lowest; FSH starting to riseEndometrium sheds with bleeding
5–13Follicular phase — dominant follicle → Graafian follicleProliferative phaseFSH and LH rise gradually; estradiol climbs and peaks just before day 14Primary oocyte completes meiosis I → secondary oocyte + first polar body
14OvulationOvulationLH surge (plus a small FSH bump); estradiol dipsGraafian follicle ruptures; secondary oocyte released, arrested at metaphase II
15–25Luteal phase — corpus luteum activeSecretory phaseProgesterone rises sharply; estradiol rises a second time; both now inhibit FSH and LHEndometrium becomes vascular, glandular, glycogen-rich for implantation
26–28Luteal regressionLate secretory → menstruationEstradiol and progesterone crashCorpus luteum dies with no LH — unless hCG from an embryo rescues it

Spermatogenesis vs oogenesis — every difference

10 rows
FeatureSpermatogenesisOogenesis
Gametes per precursor cell4 functional sperm1 ovum + up to 3 polar bodies
CytokinesisEqualUnequal — the oocyte keeps almost all the cytoplasm
TimingContinuous from puberty through most of lifeArrested twice; one oocyte finishes per cycle
First arrestNonePrimary oocyte in prophase I from before birth until ovulation
Second arrestNoneSecondary oocyte at metaphase II until fertilization
Meiosis II triggerAutomaticFertilization itself
SupplySpermatogonia self-renew — never runs outAbout 1 million follicles present at birth; the supply is fixed
Output rate100–200 million per dayNormally one egg per 28-day cycle
Support cellsSertoli cells (nurse) and Leydig cells (testosterone)Granulosa cells (estradiol)
SiteSeminiferous tubules of the testisOvarian follicles

Semen — who contributes what

4 rows
SourceShare of volumeWhat it adds
Seminal vesicles (paired)60%Fructose-rich fluid — fuel for sperm
Prostate gland30%Milky alkaline fluid
Bulbourethral (Cowper’s) glands10%Lubricates the urethra and penis tip before intercourse
Sperm themselves~1%~300 million cells in a 2–5 mL ejaculate; under 20 million/mL is considered sterile

Birth control methods (Table 51.2) — failure = pregnancies per 100 users per year

11 rows
MethodMechanismFailure rateKey drawback
ImplantHormone capsule under the skin blocks ovulation0.05Irregular or absent periods; minor surgery to place and remove
Sterilization (vasectomy / tubal ligation)Cuts and ties the vas deferens or Fallopian tubesNearly 0Reversal is expensive and often unsuccessful; rare spontaneous reconnection
IUDPrevents implantation by irritation (copper or hormonal)1A third of users get cramps or bleeding; infection risk at insertion
InjectableHormone shot every 3 months blocks ovulation6Occasional heavy menstrual bleeding
Oral contraceptiveProgesterone analogs ± estrogen suppress FSH/LH → no ovulation9 typical, ~1 perfect useMust be taken daily; risky for smokers over 35
DiaphragmRubber cup covers the cervix and holds spermicide12Needs fitting; can dislodge
CondomSheath collects semen18 (3–20 range)Needs cooperation and consistent use — but gives STD protection
Spermicides (foams, jellies, suppositories)Chemically kill or block sperm10–28Must be applied 5–10 min before each act; messy
Morning-after pill~50× estrogen dose; blocks ovulation, fertilization, or implantation1–10% per useEmergency use only — high dose, severe side effects possible
DouchingRinsing the vagina after intercourse~40Can push sperm further into the uterus — actively counterproductive
AbstinenceNo intercourse0 in principleHardest method to sustain

Exam traps

11 pairs

Questions get built out of near-misses. If you can state each difference in one sentence, you will not lose those points.

*FSH*vs*LH*

FSH = Follicle growth and Sertoli cells. LH = Leydig cells and the Luteinizing surge that causes ovulation and makes the corpus Luteum. In both sexes FSH builds gametes and LH drives the steroid hormone.

*Ovarian* cyclevs*Uterine* cycle

Two names for the same 28 days seen from two organs. Ovarian = follicular / ovulation / luteal (what the ovary is doing). Uterine = menstrual / proliferative / secretory (what the lining is doing). Follicular pairs with proliferative; luteal pairs with secretory.

*Spermatogenesis*vs*Oogenesis*

Four equal sperm, continuous, never arrests. One egg plus three polar bodies, unequal division, arrested at prophase I until ovulation and again at metaphase II until fertilization.

*Graafian follicle*vs*Corpus luteum*

Same structure, before and after ovulation. Before: a fluid-filled follicle holding the oocyte, driven by FSH, secreting mostly estradiol. After: the ruptured shell converted by LH, secreting estradiol AND progesterone.

Estradiol as *positive* feedbackvsEstradiol as *negative* feedback

Follicular phase: rising estradiol stimulates the pituitary to fire the LH surge. Luteal phase: estradiol plus progesterone together inhibit FSH and LH. Same hormone, opposite sign, different phase — this is the classic exam trap.

*Primary* oocytevs*Secondary* oocyte

Primary = diploid, arrested in prophase I, sits inside the follicle for decades. Secondary = haploid, arrested at metaphase II, and it is the cell that actually gets ovulated. A true "ovum" only exists after fertilization.

*Ovoviviparity*vs*Viviparity*

Both keep the young inside and give live birth. The difference is the food source: ovoviviparity feeds the embryo on yolk; viviparity feeds it from maternal blood through a placenta.

*Estrous* cyclevs*Menstrual* cycle

Estrous mammals reabsorb the lining (no bleeding) and are receptive only around ovulation. Humans and apes shed it with bleeding and can be receptive any time. Proestrus/estrus/metestrus/diestrus map to proliferative/midcycle/secretory/menstrual.

*Protogyny*vs*Protandry*

Protogyny = "first female", so female → male (bluehead wrasse). Protandry = "first male", so male → female. The prefix names what the animal starts as.

*Sertoli* cellsvs*Leydig* cells

Sertoli sit inside the tubule wall, respond to FSH, nurse the sperm, and secrete inhibin. Leydig sit between the tubules (interstitial), respond to LH, and secrete testosterone.

*Vasectomy*vs*Tubal ligation*

Vasectomy cuts the vas deferens — sperm are still made but never enter semen. Tubal ligation cuts the Fallopian tubes — ovulation still happens but the oocyte cannot reach the uterus. Neither removes a gonad and neither changes hormone levels.